CHEST DRAINAGE AS A THERAPEUTIC INTERVENTION
Thoracic System Pathology
When air or fluid enters the pleural space, it: 1)
separates the visceral pleura from the parietal pleura,
thus disrupting the negative pressure that prevents
the lungs from collapsing at the end of exhalation,
and 2) compresses the lung. If only a small amount
of air or fluid is present, it may be reabsorbed without
intervention. However, if large enough, the fluid or air
compromises normal respiration and must be
evacuated from the pleural space.

PNEUMOTHORAX

Air in the pleural space is called a pneumothorax,
which can be classified as spontaneous or traumatic.
Dyspnea and chest pain are the most common
symptoms of pneumothorax. Decreased breath
sounds and lack of movement on the affected side
may also be observed.

SPONTANEOUS PNEUMOTHORAX

Spontaneous pneumothorax is usually caused by the
rupture of a small bleb (enlarged air sac) on the
lung’s surface (figure 8). It typically occurs in tall, thin
men who smoke where mechanical stresses at the
apex (top) of the lung weaken the lung tissue. It may
also result as a complication of pre-existing lung
disease that weakens the lung, making it more prone
to rupture. Common causes include chronic
obstructive pulmonary disease, cystic fibrosis,
necrotizing pneumonia and AIDS patients with
Pneumocytis carinii infection.

TRAUMATIC PNEUMOTHORAX
Traumatic pneumothorax may result from:

1) Internal trauma, such as rib fracture, where the rib
punctures the lung (figure 9). If there is no opening
to the outside of the chest wall, it is called a closed
pneumothorax.

2) External trauma, such as a stab wound or bullet
wound, that penetrates the chest wall and may
puncture the lung (figure 10). This is called an open
pneumothorax or a sucking chest wound.

3) Invasive or therapeutic procedures, such as
transthoracic needle aspiration, subclavian needle
stick or thoracentesis, that inadvertently puncture the
lung. This is called iatrogenic pneumothorax. The
use of positive end-expiratory pressure (PEEP) with
mechanical ventilation can also result in iatrogenic pneumothorax, particularly in patients with acute
respiratory distress syndrome (ARDS) where the lung
tissue is weakened.

TENSION PNEUMOTHORAX

Tension pneumothorax occurs when air accumulates
in the pleural space more rapidly than it can be
evacuated. Pressure builds up which not only collapses
the lung, but can also shift the mediastinum and
severely impede venous return and cardiac output
(figure 11). A tension pneumothorax quickly becomes
life-threatening and must be relieved promptly.
Signs/symptoms of tension pneumothorax include:
rapid, labored respirations; tachycardia; cyanosis;
hypoxemia; and sudden chest pain that extends to
the shoulders. In a mechanically ventilated patient,
the high inspiratory pressure alarm may also sound.
Tracheal deviation (the trachea is skewed away from
the side of the tension pneumothorax) signifies that a
mediastinal shift has occurred and is an ominous
sign.

PLEURAL EFFUSION

Fluid in the pleural space is called pleural effusion.
The fluid may be lymph (chylothorax), pus
(empyema), blood (hemothorax), or non-specific
serous fluid. The mechanism of distress in pleural
effusion is direct compression of lung tissue; the fluid
occupies space the lung would usually fill (figure 12).
On examination, you would detect muffled or absent
breath sounds and dullness to percussion.

HEMOTHORAX

Hemothorax is defined as the presence of blood in
the pleural space. This is caused primarily by chest
trauma where virtually every blood vessel in the chest
can bleed into the pleural space. Iatrogenic
hemothorax may occur, mostly as a complication of
a central venous catheter placement.

CARDIAC TAMPONADE

Blood may also accumulate in the mediastinum,
specifically the pericardial sac, after cardiac surgery.
Accumulation of fluid around the heart can cause
cardiac tamponade. Because it compresses the heart
and interferes with venous return, it can be
life-threatening. Signs reflect the decreased venous
return: cardiac output drops severely, jugular veins
distend, pulmonary artery (PA) pressures increase,
central venous pressure (CVP) increases, and blood
pressure falls. These are ominous signs and require
immediate emergency actions.
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